herpes increased risk for hiv ?

[Herpes go away]

hello everyone.. I know herpes increases the risk of acquiring hiv so I've been diligent and have always used condoms for the peace of my mind.. but now I came across a research saying that healed skin can also favour hiv transmission so my question is even if I used condoms will there be a risk for hiv in the absence of sores ? my infected area is at the base of my penis where condoms don't cover so the vaginal fluids may come into contact with them .. I've spoken to the experts but they seem to give inconclusive answers to this question.. 

[Evaluate]

Are you having sex with girls you don't know well?  If your partner does not have HIV then you will not acquire it from them, regardless of what steps you take or don't take.  My understanding from reading literature is to do with openings in the skin, that allow bodily fluid more access to the blood stream, caused by herpes.  Even in the absence of an outbreak there can be micro sores, so to speak, that even though they may not be noticeable are still there.

[Sanguine108]

yeah, while catching HIV may be enhanced, I think what these studies are actually saying is that people who are persistently promiscuous, engage in high risk activities/decisions and are more likely to catch both HIV and HSV.

 

[dont quit!17]

http://www.medscape.com/viewarticle/472293_3

"The biology of herpetic infections supports the epidemiologic observations regarding an association between HSV-2 and HIV-1 acquisition. In experimental animal models, mucosal disruption leads to increased susceptibility to HIV-1, as measured by infection occurring at a lower inoculation of simian retrovirus infection.^[43] In addition to mucosal disruption, herpetic ulcerations are associated with an influx of large numbers of activated CD4-bearing lymphocytes,^[44,45] providing increased numbers of target cells for HIV-1 attachment and entry in the genital tract during HSV-2 reactivation. Both mucosal disruption and the presence of increased numbers of activated CD4 cells increase the likelihood that any potential exposure to HIV-1 will result in infection of the exposed host.

Several HSV proteins (ICPO, ICP27, Us11, and others) increase expression of HIV-1,^[46-49] and HSV infects and replicates in activated CD4 cells and macrophages.^[50] Both heat-inactivated and infectious HSV-1 and HSV-2 virions have been shown to increase HIV-1 expression in macrophages, likely through stimulation of nuclear factor (NF)-κ B activity.^[51] Biopsy samples have demonstrated viruses in both keratinocytes and macrophages by electron microscopy.^[52] Schacker et al.^[53] demonstrated HIV-1 RNA in 25 of 26 genital herpes lesions. HIV-1 RNA often exceeded 10,000 copies/mL of swab fluid and the quantity of HIV-1 in genital samples often exceeded HIV-1 RNA levels in blood. Similar findings were reported by investigators in India that demonstrated HIV-1 in genital ulcers in 34% of consecutive HIV-1-seropositive patients with GUD."