So I went
I apologize, this maybe a duplicate of my other question. But I'm really concerned on what is happening.
Since my first ob that consisted in tiny widespread blisters on glans and (not sure if herpes sores but) two behind leg and two on arm, I feel like my glans never returned to its original skin.
What I can see (sometimes most visible on erection) are tiny skin bumps. It feels like I'm always on outbreak, is this even possible?
They look like these (pics are not mine, just found them on internet searching "herpes glans" and they describe how my glans looks right now). Could herpes give status of constant tiny bumps in people with low body defence? And since I have them, means I'm constantly shedding?
Thanks for your opinions!
(I am tested HSV2 positive with value 1, three months after exposure)
Hello friends, my IGG is increasing I'm now 1 at blood test, was 0,50 after my glans became... dotted. If you want read my story I'm one of those unlucky (maybe 'cause my body defences sucks?) got it from an oral received (sex worker).
I'm in the guess usual phase of depression and thoughts as you can imagine and know.
I have few questions:
Feel like my glans is not anymore like used to be before the outbreak (my ob looked more like a balanitis, never scab, only widespread tiny pimples), I can still see tiny skinbumps... maybe on my body I'll have a costant mini outbreak like this (I'm now 3 months half after intercourse)? Am I condamned to never give orals to girls anymore? Anyone here considered to joing sperimental hsv-2 cure programs if they exist? Thanks in advance
For those with questions related to IgM and IgG antibodies and the body’s immune responses here is an explanation an actual Herpes researcher himself (whose name I will not reveal) so new members are not stuck in the dark with explanations from unqualified members:
IgM is produced upon primary exposure, but those wane and never come back. IgG is produced after what's called class switching, and appears late in the first response (at extremely low levels), then higher and higher levels upon each subsequent exposure. If there is no exposure for a long time, the levels of IgG drop and are not protective. The best example of this is the need for regular tetanus booster shots; if you are not exposed to tetanus toxin between boosters, the protective level of IgG produced from the vaccine drop to a non-productive level; the booster gives the immune system a reminder and gooses up the IgG level to be protective again. In the case of herpesviruses, if the virus remains latent for a very long time, whatever protective immune reaction you had to it can wane and no longer be protective when a OB now occurs.
Bottom line: primary exposure to anything (vaccine, first time your seeing an infectious agent, allergen, etc.) will generate an IgM response. Those B cells that produced IgM will go into memory, where they will be available to respond to a second or subsequent exposure. If the same antigen is encountered again, genetic changes take place in those memory cells that allow them to switch to producing IgA, IgG, and/or IgE, all of which are more effective for a protective response. If the stimulus reappears (usually we're talking 6-8 week increments between exposures), the IgG/IgE/IgA response is boosted even more; essentially your immune system is being told that whatever this antigen is, you keep getting exposed to it and the previous level of protection was clearly not enough; we gotta do more.
If the stimulus is cleared and not encountered again, eventually the level of protective antibodies will wane; it's a way for your body not to waste valuable energy resources on something it doesn't need. Hence the need for periodic booster shots; you remind your waning protective response that this agent needs a higher level of antibodies to be protected. By reintroducing your immune system to the antigen, you're telling your immune system to boost its IgG/IgA/IgE against it.
The way this applies to herpes is that your first antibody response from primary exposure will ALWAYS be an IgM response. However, that it frequently overlooked because it can happen even in the absence of signs or symptoms. Each OB acts like a stimulation, leading to the class switch to IgG or IgA antibodies against the virus. Each measurement of the antibody level, though, is a snapshot in time; one cannot tell when or how stimulated the immune system was. One must follow the course of the antibody levels. If they are waning over time, then the infection has already passed and your body is winnowing out unneeded antibodies (and cells producing those antibodies). If the levels are waxing, then you were recently re=stimulated to produce antibodies. If the levels are staying the same, there's not much one can say except you've had, or are having, an immune stimulus.
Hello heres my situational
I have been diagnoses with genital hsv1 my parther has oral hsv1 we have and had unproteced sex with no active lession what are chances of me transmitting to her genital does having the same hsv1 diganoses but in differnt areas mean that antibody with proteced the rest of body? Any one else in a simlair siuational? With a parther having the same virus but in a different location?
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